CJC-1295 with DAC peptide lyophilized powder 5mg

CJC-1295 with DAC peptide lyophilized powder 5mg

Product Name：CJC-1295

CAS：863288-34-0

Appearance：white lyophilized powder

Specification：per vial 5mg

Package：10 vials/box

Contact: Liu Zheng

WhatsApp/Telegram: +86 17732181778

Email: liuzheng@hnttmy.cc

We can provide all kinds of peptides：Tirzepatide，Retatrutide，Semaglutide，GLP-1，NAD/NAD+，GHK-CU，BPC-157，TB500，MOTS-C，AOD 9604，CJC-1295，Glutathione，Cagrilintide，Melanotan II，IGF-1 LR3，Tesamorelin，Ipamorelin，HCG，HGH，Somatropin，Dihexa，etc hot selling peptide and Bacteriostatic water.

Nicotinamide adenine dinucleotide participates in a variety of physiological activities such as cell metabolism, energy synthesis, and cell DNA repair, and plays an important role in the body's immune ability.

In a healthy state, the concentration of nicotinamide adenine dinucleotide in the human body is stable, maintaining the normal functions of various cells. The concentration of nicotinamide adenine dinucleotide in the body determines the process and degree of cell aging, and a decrease in concentration will accelerate the process of cell aging.

Studies have shown that NAD+ has a protective effect on renal infarction caused by ischemic surgery, and can significantly reduce the levels of urea nitrogen and creatinine in serum; NAD+ has a protective effect on renal tubular damage caused by ischemic surgery. NAD+ (Nicotinamide adenine dinucleotide Chemicalbook acid, Nicotinamide adenine dinucleotide, referred to as NAD+) can effectively protect renal damage caused by renal ischemia, and NAD+ has important application value in the preparation of drugs for the prevention and treatment of renal ischemic damage.

In addition, nicotinamide adenine dinucleotide has certain applications in the preparation of drugs for the treatment of inflammatory pain. NAD+ is involved in regulating formalin and complete Freund's adjuvant (CFA)-induced inflammatory pain through the NAD+-dependent deacetylases SIRT1 and SIRT2, while SIRT1 and SIRT2 are involved in the inhibitory effect of NAD+ on inflammatory pain through different mechanisms, thereby achieving analgesic effect on inflammatory pain.

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